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All the patients got the BRAF mutation. Dr Chapman stated: ‘Why is this treatment not the same as regular chemotherapy is that regular chemotherapy episodes the machinery involved in cell division; so to stop the cancer cells dividing uncontrollably, most standard chemotherapy aims to block the mechanism of division by interfering directly with DNA replication or with microtubules in the dividing cells. PLX4302 differs because it episodes the genetic programme that is causing the cells to divide uncontrollably, and the BRAF is believed by us mutation is driving that programme. The drug is certainly blocking the genetics of the tumour, rather than trying to hinder the proliferation of the cells and, as a total result, there are fewer unwanted effects, although there are some.